“Maternal instinct, ought to have been the driving force concerning use of marijuana and the potential congenital damage to the health of the fetus, but thanks to our media, it’s largely been ignored.
Regarding the now well-accepted effects of consumption during adolescence: the uniform voice of all the respiratory physicians, the long term brain structure changes, the high incidence of psychiatric disorders, and the now proven gateway effect, one must realize there is quite a unison chorus, all crying “caution.”
Legalizers, of course, with the help of the media, hide the truth! The debate at present, relates to “civil rights” – the users right to get stoned without affecting anybody else – or at least, doing minimal harm, to those around them.
They not only ignore the work of the eminent Prof. Nahas, but in light of their goal, full legalization, becoming honest about the dangers, is just not going to happen.
But we too, must accept our part in this failure – the ‘follow-up’ on the vital dissemination of this information. Rather, lies and propaganda “linger-lacking” waiting, as government ignores it’s primary mandate – to protect the populace – and the legalizers, of course, hire pseudo-scientists, with a goal of validating any imagined implication of medicinal use!
Congenital effects: has the alarm been sounded loudly enough?
In his book, “Keep Off the Grass,” published some 28 years ago, Gabriel G. Nahas, M.D., Ph.D., devoted an entire chapter to “Marijuana and DNA” in which he described experiments he and his fellow researchers noted abnormalities in the biology of the human cell, specifically lymphocytes, as a result of smoking marihuana.
When these cells were cultured for three days, the ’cells from the marihuana smokers showed “an abnormally high number of chromosome breaks” compared with cells taken from non-smokers.’
Later in his book, when hypothesizing about possible fetal effects of marihuana smoking, Nahas stated:
“From my own observations, it is logical that a substance known to slow down the division of cells could affect the fetus, an organism in which cell division is occurring at a very rapid rate.”
Parallels with tobacco consumption, also ought to have been a red flag!
The majority, now aware, of course, that tobacco has been definitively linked with lung cancer, few are cognizant of the dose-response – relationship between lifetime tobacco consumption – the interaction of the dose and duration. In recent decades, it has been demonstrated with the lung cancer incidence.
But what is not widely understood is that the best relationship was shown by advanced U.N. statisticians to be with the FOURTH POWER OF THE DURATOIN OF TOBACCO EXPOSURE – so that if one smokes tobacco for twice as long then one’s incidence of lung cancer rises 16-FOLD!
But when we factor in the congenital abnormalities – the whole discussion is re-cast completely.
All studies show increased:
cono-ventricular septal defects
greatly increased gastroschisis,
8 fold elevated anotia (born with no ears) or microotia;
and neural tube closure defects (spina bifida etc.).